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In humans, IGF-I also increases renal blood flow and GFR by ~25% and causes volume expansion and sodium retention by a direct action on the renal tubules, with stimulation of rennin release and suppression of atrial natriuretic peptide secretion[364]

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Transferred mitochondria accumulate reactive oxygen species, promoting proliferation

doi: 10.1097/MEG.0000000000001987 Sergio Cadoni, Sauid Ishaq, Cesare Hassan, Pemysl Falt, Lorenzo Fuccio, Keith Siau, Joseph W Leung, John Anderson, Kenneth F Binmoeller, Franco Radaelli, Matt D Rutter, Shinya Sugimoto, Humayun Muhammad, Pradeep Bhandari, Peter V Draganov, Piet de Groen, Andrew Y Wang, Andrew W Yen, Chris Hamerski, Henrik Thorlacius, Helmut Neumann, Francisco Ramirez, Chris J J Mulder, Eduardo Albniz, Arnaldo Amato, Makoto Arai, Adrian Bak, Maximilien Barret, Putut Bayupurnama, Ramsey Cheung, Hey-Long Ching, Hartley Cohen, Sunil Dolwani, Shai Friedland, Hideaki Harada, Yu-Hsi Hsieh, Bu Hayee, Toshio Kuwai, Vicente Lorenzo-Ziga, Mauro Liggi, Takeshi Mizukami, Donatella Mura, David Nylander, Snorri Olafsson, Silvia Paggi, Yanglin Pan, Adolfo Parra-Blanco, Rupert Ransford, Joaqun Rodriguez-Sanchez, Hakan Senturk, Noriko Suzuki, Chih-Wei Tseng, Hugo Uchima, Noriya Uedo, Felix W Leung